Light has played a crucial role in shaping the behavior and physiology of most species on Earth. For diurnal animals, including humans, natural daylight has been a key regulator of wakefulness, while the onset of darkness signals the time for sleep. This light–dark cycle has been a constant throughout most of our evolutionary history. However, the advent of artificial light has dramatically altered these natural patterns, extending human activity into the night and giving us control over when and how we engage with our environment. While these advancements have brought about numerous benefits, they have also introduced significant challenges to our health.
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One of the most critical issues arising from the use of artificial light is its impact on sleep and circadian rhythms. The human circadian rhythm, which regulates our sleep-wake cycle, is highly sensitive to light. Disruptions in this rhythm due to irregular light exposure have been linked to various health problems, including sleep disturbances and an increased risk for obesity and metabolic disorders. These conditions have been on the rise globally, partly due to our altered light exposure patterns.

Light is detected by specialized photoreceptors in the retina, including rods, cones, and intrinsically photosensitive retinal ganglion cells (ipRGCs). While rods and cones are primarily responsible for image formation, ipRGCs play a crucial role in non-visual responses to light, such as circadian entrainment and sleep regulation. These cells are particularly sensitive to light at around 480 nm (blue light), which is known to have strong effects on circadian rhythms. The ipRGCs transmit light information to the brain's central circadian pacemaker, the suprachiasmatic nucleus (SCN), which regulates the release of melatonin from the pineal gland. Melatonin is a key hormone that promotes sleep and helps maintain the circadian rhythm by signaling the body when it’s time to sleep.

Beyond sleep regulation, light exposure also influences energy metabolism. During the deepest stage of non-rapid eye movement (NREM) sleep, known as slow-wave sleep (SWS), energy expenditure is at its lowest, indicating that sleep serves as a period of energy conservation. The SCN also drives daily rhythms in the concentrations of various hormones linked to metabolism, such as insulin, glucagon, and corticosterone, which in turn influence energy balance and substrate utilization in the body.
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The disruption of natural light cycles, particularly through exposure to artificial light at night, can therefore have wide-ranging effects on both sleep and metabolic health. Understanding these mechanisms is critical as we navigate the modern world, where artificial light is ubiquitous and often unavoidable. Balancing our exposure to natural and artificial light may be key to maintaining optimal health and well-being.

The central circadian clock in the SCN is highly sensitive to external light cues, with characteristics such as intensity, duration, timing, and wavelength playing critical roles in regulating sleep and circadian rhythms. Each of these light properties has specific effects on human physiology, with varying degrees of influence depending on their combination and context.

Natural light intensities can range dramatically, from the intense midday sunlight of 20,000 to 100,000 lux to the much dimmer artificial indoor light, which typically ranges between 14 and 430 lux. Human exposure to these varying light levels can significantly influence physiological processes, especially when these exposures occur at night.

Studies have shown that increased light intensity at night can disrupt sleep and shift circadian rhythms. For example, continuous illumination during the dark phase in mice has been linked to increased body mass and reduced glucose processing, even without changes in food intake or activity levels. Similarly, in humans, increasing light intensity at night can lead to melatonin suppression and increased alertness, indicating a phase shift in the circadian rhythm. Epidemiological studies have also documented a relationship between light intensity at night and metabolic health. For instance, brighter bedroom environments have been associated with a higher risk of obesity and metabolic disorders, such as diabetes.

Moreover, even low levels of light at night (as low as 5 lux) can disrupt sleep architecture, leading to changes in sleep stages and a decrease in total sleep time. This disruption in sleep has secondary effects on metabolism, potentially increasing the risk for insulin resistance and other metabolic issues.

In addition to intensity, the duration and timing of light exposure are crucial. Prolonged exposure to artificial light at night can exacerbate its disruptive effects on sleep and circadian rhythms. The timing of light exposure is equally important, with evening exposure being particularly detrimental. Late-day light exposure can delay the onset of melatonin secretion, shifting the circadian rhythm and leading to difficulties in falling asleep and maintaining sleep.

Furthermore, light exposure in the evening has been shown to increase the risk of metabolic disorders. For instance, studies have found that individuals exposed to light in the evening are more likely to develop insulin resistance, which can lead to type 2 diabetes. This effect is partly mediated by the impact of light on the sympathetic nervous system, which regulates glucose metabolism.

While intensity and duration are significant, the wavelength or spectral composition of light also plays a critical role. Blue light, with a wavelength around 480 nm, is particularly effective at suppressing melatonin and disrupting sleep. This has implications for the use of screens and devices that emit blue light, especially in the hours leading up to bedtime.
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Other characteristics, such as the color temperature of light, flickering, and the type of light source (e.g., LED vs. OLED), also influence physiological responses. For instance, light-emitting diodes (LEDs) often have higher blue light content compared to organic light-emitting diodes (OLEDs), making them more likely to disrupt sleep and circadian rhythms.

The duration of light exposure significantly affects sleep and circadian rhythms in a dose-dependent manner. Research has shown that even a single session of high-intensity light exposure can have profound effects. In a study involving 39 healthy young adults, exposure to 10,000 lux light for varying durations (ranging from 0.2 to 4.0 hours) led to dose-dependent suppression of melatonin and shifts in circadian rhythm. This finding underscores the sensitivity of the circadian system to prolonged light exposure, particularly at high intensities.

The effects of light exposure duration extend beyond circadian disruption and impact metabolic health as well. For instance, in a study of 48 young children, prolonged exposure to light above 200 lux was associated with increased body mass index (BMI), even when controlling for sleep duration, timing, and activity levels. Additionally, observational studies have linked extended screen time, a form of prolonged light exposure, to an increased risk of overweight and obesity in children and adolescents. A meta-analysis further supported this association, highlighting the metabolic consequences of extended exposure to light from electronic devices.

The nature of light exposure, however, is complex. Circadian phase shifts can occur even with brief, intermittent light exposure. In one study, as little as 60 minutes of 2-millisecond light pulses in the evening led to significant phase delays in the circadian rhythm of participants. This finding suggests that not only continuous light exposure but also intermittent light patterns can disrupt circadian timing and potentially affect metabolism.

The timing of light exposure plays a crucial role in determining its impact on the circadian system. Depending on whether light exposure occurs in the early or late evening, the circadian rhythm may either advance or delay. This has important implications for sleep quality and metabolic regulation. For example, exposure to light in the late evening is more likely to phase delay the circadian system, leading to difficulties in falling asleep and potential metabolic disruptions.
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In summary, both the duration and timing of light exposure are critical determinants of their effects on sleep and metabolism. Prolonged and mistimed light exposure, whether continuous or intermittent, can lead to circadian misalignment and adverse metabolic outcomes. Understanding these dynamics is essential for mitigating the potential negative health effects of artificial light in modern environments.

​Morning bright light exposure is widely recognized as an effective treatment for individuals with Seasonal Affective Disorder (SAD) and winter depression, helping to shift the circadian rhythm and potentially improve metabolic states. Research has shown that such exposure can lower the resting metabolic rate (RMR) in SAD patients and may lead to reductions in body weight and depressive symptoms. Studies also indicate a potential influence on glycemic control, as observed in diabetic patients with winter depression, though the effects on metabolism in individuals without depression are less consistent. The combination of morning light therapy and exercise has shown promise in reducing body fat among overweight individuals. Additionally, morning bright light is effective in improving sleep, making it a viable non-invasive treatment for circadian rhythm and metabolic disorders. Further research is needed to explore these benefits across different populations.

Daytime light exposure in animals is often considered a “dead zone” in the circadian phase-response curve, where light does not significantly reset the circadian rhythm. The presence of this "dead zone" in humans remains uncertain due to differences in activity patterns between diurnal and nocturnal species. Few studies have explored how daytime light exposure impacts human metabolism.

Research shows mixed results: 14 hours of daytime light exposure did not significantly affect 24-hour energy expenditure or fat and carbohydrate oxidation in healthy individuals. However, dim daytime light combined with bright evening light reduced the usual rise in postprandial glucose in insulin-resistant older adults. Additionally, daytime light treatment in individuals with SAD led to weight loss and increased oxygen consumption. Dim light during the day also affected digestion, leading to signs of carbohydrate malabsorption and reduced gastric activity.
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Daytime light exposure can also influence metabolism during sleep, with studies indicating that dim daytime light and bright evening light may decrease the sleeping metabolic rate. These findings highlight the significant role of daytime light conditions in sleep and overall energy metabolism, warranting further investigation into their long-term health implications.

Extended light exposure during the dark phase can significantly disrupt metabolism. In animals, constant light exposure reduces the amplitude of the circadian rhythm in the suprachiasmatic nucleus (SCN), increases food intake, decreases energy expenditure, and leads to weight gain and reduced insulin sensitivity. This exposure disrupts the regular circadian rhythm and peripheral clocks, contributing to metabolic imbalances.
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In humans, evening and prolonged light exposure are linked to higher body weight, increased BMI, and a greater risk of obesity. Actigraphy studies have shown a positive correlation between mean light exposure timing, BMI, and sleep midpoint, indicating the role of light in metabolic regulation.
Shift workers, who are exposed to light during atypical hours, consistently show higher risks of metabolic disorders, including overweight, obesity, diabetes, and metabolic syndrome. This population often faces circadian disruption, sleep deprivation, and irregular eating patterns due to their work schedules, further complicating metabolic health.

Evening light exposure also affects energy metabolism during sleep. Studies have shown that exposure to bright light before sleep increases respiratory quotient and decreases fat oxidation, suggesting a shift towards carbohydrate metabolism. This is accompanied by a suppression of melatonin, a hormone crucial for regulating sleep and metabolic processes.

Furthermore, evening light exposure can impair carbohydrate digestion and increase glucose intolerance and insulin insensitivity, especially when combined with disrupted sleep and circadian misalignment. Shift workers and individuals with night eating syndrome are particularly vulnerable to these metabolic disturbances, often showing a preference for high-fat foods and altered dietary intake patterns.

Understanding the timing and intensity of light exposure, along with dietary habits, is essential for minimizing metabolic consequences, particularly in populations with atypical light exposure, such as shift workers. Proper management of light exposure and food intake timing can help maintain circadian rhythm and support healthier metabolic outcomes.

In humans, the activity of the suprachiasmatic nucleus (SCN) is often gauged through endogenous melatonin levels, with nighttime production typically ranging from 10 to 80 μg in young adults. Peak melatonin concentrations vary widely, with one study reporting levels between 2 and 84 pg/mL among a group of 170 individuals. Although melatonin secretion is primarily driven by photic input, its receptors are distributed throughout the body in areas beyond the pineal gland, such as the retina, gastrointestinal tract, bone marrow, skin, and lymphocytes. This widespread distribution means that melatonin’s influence extends beyond regulating sleep and circadian rhythms, playing significant roles in thermoregulation and energy metabolism.
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Research has demonstrated the critical role of melatonin in metabolic processes. Animal studies involving pinealectomy, which removes the source of melatonin, have shown that the absence of melatonin leads to metabolic abnormalities, including diminished glucose tolerance, reduced glycogen storage in the liver and muscles, and insulin resistance—conditions that mirror those found in diabetogenic syndrome. In humans, similar findings have been observed, with reduced melatonin amplitude and blunted rhythms reported in individuals with type 2 diabetes. Additionally, melatonin interacts with insulin, particularly in individuals with metabolic syndrome, highlighting its vital role in maintaining energy balance and metabolic health.

Exogenous melatonin has been widely studied for its effectiveness in managing sleep disorders and circadian rhythm disruptions, particularly in individuals experiencing jet lag, shift work, or visual impairments. Earlier research has established its role in improving sleep quality and aligning circadian rhythms in these populations. Beyond its influence on sleep, melatonin has garnered attention for its potential effects on human metabolism, particularly in the regulation of lipid and glucose metabolism.

In women with obesity, studies have highlighted a negative correlation between melatonin supplementation and BMI, suggesting potential benefits for weight management. For instance, a three-week randomized crossover trial involving individuals with type 2 diabetes and insomnia revealed that melatonin treatment improved sleep efficiency and reduced wakefulness after sleep onset, though it did not significantly impact glucose or lipid metabolism. Another study focusing on normolipidemic postmenopausal women found that a two-week course of melatonin (6 mg nightly) led to an increase in plasma triglyceride and VLDL cholesterol levels, underscoring the hormone's complex metabolic effects.

The benefits of melatonin extend to the shift-working population, where its administration has been shown to alleviate circadian misalignment and enhance sleep quality, alertness, and energy intake. Notably, a randomized crossover trial demonstrated that melatonin (3 mg) reduced body weight, BMI, waist circumference, and hip circumference in shift workers without altering caloric intake, alongside a significant reduction in circadian misalignment. Another trial over 12 weeks in female shift workers with elevated BMI reported that melatonin administration did not significantly affect energy intake or food choices, indicating that melatonin's effects on weight may be independent of dietary factors.

Interestingly, the interaction between melatonin and light exposure has also been explored, revealing varying effects on metabolism. For example, in a study involving healthy males, nighttime melatonin administration under bright light conditions increased leptin levels and reduced hunger, along with improvements in glucose tolerance and insulin sensitivity. However, contrasting findings were observed in healthy females, where melatonin impaired glucose tolerance, suggesting a potential decrease in insulin sensitivity. These mixed results, likely influenced by differing light conditions across studies, highlight the need for future research to clarify the interplay between melatonin and environmental light.

In summary, while exogenous melatonin shows promise in improving sleep and potentially influencing metabolic outcomes, its effects are nuanced and may vary based on individual factors such as light exposure and underlying metabolic conditions.

Melatonin, a hormone that follows a daily rhythm in vertebrates, also exists in various non-animal sources, such as unicellular algae, food plants, and medicinal herbs. This naturally occurring melatonin can be found in fruits, vegetables, grains, and beverages like coffee, tea, beer, and wine. Some foods, particularly cranberries, coffee, and certain herbs, are known to contain high levels of melatonin. Additionally, melatonin is present in meats like lamb, beef, pork, chicken, and fish. However, the impact of melatonin from these food sources on human physiology, particularly sleep, remains an area of ongoing research, as the concentration of melatonin in foods can vary significantly.

Fruits such as sour cherry, also known as Montmorency cherry, are particularly noted for their high melatonin and tryptophan content, both of which are linked to improved sleep. In studies involving healthy adults, consuming tart cherry juice for a week significantly increased melatonin levels and improved sleep parameters, including total sleep time and sleep efficiency. The sleep-promoting effects of cherry-based products have been further explored in older adults and individuals with insomnia, where improvements in sleep duration, sleep quality, and reduced wakefulness after sleep onset were observed. Moreover, studies on fruits like pineapple, oranges, and bananas have demonstrated elevated serum melatonin concentrations following their consumption, suggesting a potential role in sleep enhancement.

Milk, which naturally contains both tryptophan and melatonin, has also been investigated for its potential to improve sleep. However, the melatonin content in milk can vary widely, making it challenging to measure its exact impact on sleep. Research involving melatonin-enriched milk has shown promising results, particularly in young adults, where significant improvements in sleep satisfaction and reductions in daytime sleepiness were noted. In children, while milk-based evening drinks did not significantly affect overall sleep time, they did reduce nocturnal awakenings and improved memory recall, indicating some benefits. The use of fermented milk with probiotics, such as Lactobacillus casei strain Shirota (LcS), has also shown to improve subjective sleep quality and reduce sleep latency under stressful conditions.

​Grains, including rice, corn, barley, and whole grains, have been identified as high in melatonin, and their consumption has been associated with better sleep quality. For example, a study on cereal enriched with tryptophan demonstrated improvements in sleep efficiency and total sleep time in older adults. The potential hypnotic effects of food-derived tryptophan have also been explored, with evidence suggesting that tryptophan-rich foods, such as de-oiled gourd seeds, can improve insomnia when combined with carbohydrates. Additionally, tryptophan supplementation, particularly when paired with daytime light exposure, has been shown to promote evening melatonin secretion and enhance sleep.

In summary, while melatonin is present in a wide variety of foods, its effectiveness in improving sleep and metabolism requires further investigation. The interaction between melatonin and other nutrients within these foods, as well as the role of environmental factors like light exposure, is complex and necessitates well-controlled studies to fully understand its impact on human health.

Ishihara, Asuka, et al. “The Complex Effects of Light on Metabolism in Humans.” Nutrients, vol. 15, no. 6, 14 Mar. 2023, pp. 1391–1391, https://doi.org/10.3390/nu15061391. 

Technologies like cellular phones and wireless devices are ubiquitous in our daily lives, serving as essential tools for communication, entertainment, and productivity. In recent years, the proliferation of these wireless internet technologies has led to the mainstream become more aware of these devices emitting significant amounts of electromagnetic radiation (EMR)/electromagnetic frequencies (EMF). These devices, which operate as radio devices transmitting and receiving radio EMF within a large band of radio frequencies (RF), come with significant health concerns, particularly in the realm of reproductive health.

​The radiation emitted by mobile phones can have both thermal and non-thermal impacts on biological materials, with potential long-term effects on cellular functions and the hormonal balance in the human body. Emerging research points to a troubling link between mobile phone usage and male infertility, a condition that already affects nearly half of the 15% of couples worldwide struggling with reproductive issues.

For years the cell phone companies have assured people that cell phones are perfectly safe. Currently there are over 700 million cell phone users in the world. Analog phones operate at 450–900 MHz, digital phones (Global System for Mobile Communications [GSM]) at 850–1900 MHz, and third-generation phones at approximately 2000 MHz. ​The radiation emitted by Wi-Fi and all generations of mobile phones is classified as non-ionizing radiation, which falls within the microwave range (3–300 GHz).

• First generation (1G) and second-generation (2G) mobile phones operate at a frequency range of 850–1900 MHz
• Third-generation (3G) mobile phones can reach up to 2,500 MHz
• The most recent fourth-generation (4G) and fifth-generation (5G) mobile technologies operate at a wider and higher frequency range of 2–8 GHz and 3–300 GHz, respectively 
• Unlicensed spectrum bands of 2.4 and 5 GHz are used by Wi-Fi devices

​5G routers and modems, operating on higher frequencies, emit more powerful electromagnetic fields, potentially amplifying the risks. The introduction of 5G technology, which involves more frequent data transmissions at higher power levels, has raised concerns about whether this could intensify the reproductive risks posed by EMF radiation.

Keep in mind, the EMFs emitted by cell phones are a form of microwave energy. Specifically, cell phones emit RF radiation, which falls within the microwave portion of the electromagnetic spectrum. Microwaves, including the frequencies used by cell phones, are non-ionizing radiation, meaning they don't carry enough energy to ionize atoms or molecules.

Cell phones typically operate at frequencies between 800 MHz and 2.6 GHz, which are in the lower part of the microwave frequency range. This type of radiation is also used in other wireless technologies, such as Wi-Fi and Bluetooth.
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While the power levels of cell phones are much lower than those of devices like microwave ovens, the concern over potential health effects has led to ongoing research on the long-term exposure to RF radiation emitted by mobile devices.

Impact on Semen
Studies examining the association between mobile phone use and semen parameters have yielded significant results. Men who stored mobile phones in their trouser pockets exhibited a decrease in the percentage of normal sperm morphology and luteinizing hormone levels. Additionally, exposure to mobile phone EMR was associated with:

• reduced sperm motility (sperm movement): For sperm to fertilize an egg, they need to be able to swim through the female reproductive tract to reach the egg. Sperm that move actively and in a straight line are considered to have good motility.
• reduced linear velocity (sperm speed): sperm with higher linear velocity are more likely to reach the egg, improving chances of conception. If sperm swim too slowly or in a disorganized way, they may not get there in time.
• reduced linearity index (how straight the sperm’s path is when it swims): A high linearity index means sperm are moving directly toward their target (the egg), which makes fertilization more likely. Sperm with a low linearity index may swim in random directions, wasting energy and reducing the chance of fertilization.
• reduced acrosin activity (ability to penetrate egg): Acrosin is an enzyme found in sperm that helps it penetrate the outer layer of the egg. Think of it like a key that helps sperm unlock the egg. Acrosin activity refers to how well this enzyme is working. Healthy acrosin activity ensures that sperm can do their job once they reach the egg.
• increased sperm DNA fragmentation (DNA damage): DNA fragmentation means that the sperm’s genetic material (DNA) is damaged or broken into pieces. The DNA inside sperm is critical because it carries the genetic information that gets passed on to the baby. Sperm with high levels of DNA fragmentation have damaged genetic material. High DNA fragmentation can lead to problems in fertilization, miscarriage, or developmental issues in the baby. Even if sperm are able to fertilize an egg, if the DNA is damaged, the embryo may not develop properly. This can reduce the chances of a successful pregnancy.

The frequency and duration of mobile phone use have been linked to declines in semen volume, sperm concentration, and total sperm count, indicating a detrimental effect on sperm quality and male fertility. Notably, carrying cell phones in hip pockets and on belts has been associated with lower sperm motility compared to other storage methods.
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Moreover, prolonged exposure to EMF from mobile phones and routers has been linked to a higher rate of childlessness among certain professions, such as military personnel in the Royal Norwegian Navy. These findings suggest that frequent exposure to mobile phone radiation may impair reproductive health over time.

​Numerous studies have shown that radiation emitted by Wi-Fi and 5G routers, especially when used for prolonged periods, can negatively affect sperm quality, including sperm count, motility, and DNA integrity.

A laboratory study found that exposing sperm samples to a laptop connected to Wi-Fi for just four hours significantly reduced sperm motility and increased DNA fragmentation. This indicates that not only direct phone use but also proximity to routers and modems could affect sperm health.

In human studies, semen analysis in the four cell phone user groups showed a decrease in sperm count, motility, viability, and normal morphology with the increase in daily use of cell phone - in a dose dependent manner (the more EMF radiation exposure, the greater the effects to semen). Other researchers suggested in their study on mice that Leydig cells are among the most susceptible cells to EMW, and injury to Leydig cells may affect spermatogenesis. Additionally, mobile phone EMR induced genotoxic effects on epididymal spermatozoa, which is critical for fertility.

Research indicates that prolonged RF-EMR exposure, such as frequent use of mobile phones over several years, can lower testosterone levels in men. Testosterone is a critical hormone for sperm production and general male health. Over time, men using mobile phones emitting 950 MHz RF-EMR experienced a gradual reduction in testosterone levels. ​

Additionally, RF-EMR negatively affects the anterior pituitary gland, which regulates several hormones, including cortisol, thyroid hormones, and adrenocorticotrophic hormone (ACTH). This interference with hormonal balance may result in decreased reproductive function.

Some studies have suggested that mobile phone radiation could lead to Leydig cell hyperplasia, a condition where these testicular cells overgrow and produce elevated testosterone levels. However, this increase is misleading, as reproductive functions, such as sperm quality, are still impaired despite the rise in testosterone. Decreased sperm count, motility, and quality have been consistently linked to mobile phone use, validating the harmful impact of mobile phone radiation on male fertility.

Animal Studies on RF-EMR Exposure
Animal studies have further validated these concerns. Exposure to RF-EMR, particularly at 900 MHz, has been shown to increase the levels of reproductive hormones such as FSH (Follicle Stimulating Hormone), LH (Luteinizing Hormone), and prolactin in animals. While these hormones are typically involved in regulating male reproductive functions, prolonged exposure to RF-EMR disrupts this balance. For example, increased LH levels in animals exposed to mobile phone radiation were accompanied by damage to Leydig cells via changes in protein kinase C, which led to reduced testosterone production.

Additionally, RF-EMR exposure increases oxidative stress in Leydig cells, leading to cellular damage and apoptosis (cell death). This oxidative stress, combined with thermal effects from radiation, can impair the function of the hypothalamus and pituitary gland, which are essential for regulating reproductive hormones like LH and FSH. When these hormones are out of balance, the entire reproductive system can be negatively impacted.

Human Studies on RF-EMR Exposure
In studies of men, the group exposed to EMFs had a considerable decrease in LH levels. Additionally, RF-EMR appears to have a negative relationship with the anterior pituitary gland
and the downstream effects of hormones released via the actions of the pituitary. Studies of men with long-term use of 950 MHz mobile phones (6 years) have revealed reduced testosterone levels, which is dependent on time, likely due to damage to Leydig cells and insufficient LH, as LH stimulates the secretion of testosterone by testicular Leydig cells.

These hormonal regulations by the hypothalamus and anterior pituitary are essential for male reproductive functions. RF-EMR emitted from mobile phones can cause thermal effects as manifested by the elevation of temperature and EMF strength value on the hypothalamus and pituitary gland after mobile phone exposure. The penetration of RF-EMR on the hypothalamus and pituitary gland is deeper in lower frequency bands (700 and 900 MHz).

Meo, Sultan, et al. Effects of Mobile Phone Radiation on Serum Testosterone in Wistar Albino Rats. 2010.

Maluin, Sofwatul Mokhtarah, et al. “Effect of Radiation Emitted by Wireless Devices on Male Reproductive Hormones: A Systematic Review.” Frontiers in Physiology, vol. 12, 24 Sept. 2021, p. 732420, www.ncbi.nlm.nih.gov/pmc/articles/PMC8497974/, https://doi.org/10.3389/fphys.2021.732420. Accessed 22 Oct. 2021.
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Okechukwu, Chidiebere Emmanuel. “Does the Use of Mobile Phone Affect Male Fertility? A Mini-Review.” Journal of Human Reproductive Sciences, vol. 13, no. 3, 2020, p. 174, https://doi.org/10.4103/jhrs.jhrs_126_19.

​Agarwal, Ashok, et al. “Effect of Cell Phone Usage on Semen Analysis in Men Attending Infertility Clinic: An Observational Study.” Fertility and Sterility, vol. 89, no. 1, Jan. 2008, pp. 124–128, https://doi.org/10.1016/j.fertnstert.2007.01.166.

It is widely recognized that radiation exposures such as X-rays and gamma radiation can increase the risk of cancer in humans and animals. These types of radiation are referred to as ionizing radiation (Ionization energy is defined as the minimum amount of energy required to remove an electron from an atom or molecule in the gaseous state). This is different from nonionizing radiation, which includes ultraviolet (UV), visible light, extremely low frequency radiation (ELF), and radiofrequency or microwave (RF) radiation. Conventionally, researchers believed that nonionizing radiation is not harmful or carcinogenic, despite evidence surfacing regarding the relationship between UV radiation and skin cancer.

Research evaluating the exposure to RF radiation was conducted primarily by military agencies. Due to the advent and use of cellular telephone systems, which involve widespread public exposures, reevaluation of exposure risk has become urgent.

Four types of physiological effects has been observed by researched in multiple studies:

1. Increased spontaneous abortion,
2. Shifts in red blood cell and white blood cell count,
3. Increased somatic mutation rates in lymphocytes, and
4. Increased childhood, testicular, and other cancers.

These findings suggest that exposure to RF radiation, including from devices such as microwave ovens, are potentially carcinogenic and have other health effects. An important point to consider is that low dose radiation exposure over time has damaging effects, rather than one large exposure to radiation.

Rather than using a microwave, consider using a stove or convection oven. These methods take a bit more time, but it is well worth it! Consider using headphones to talk on cell phones rather than placing the device directly to your head. If possible, request to avoid airport scanners by opting for a pat-down. Lastly, try to reduce overall radiation exposure over time.

Goldsmith, J. (1997). Epidemiologic Evidence Relevant to Radar (Microwave) Effects. Environmental Health Perspectives, 105, 1579. https://doi.org/10.2307/3433674

The environment that we live in is toxic. It is worrisome to think that the status quo has occurred with the help of corporations knowingly dumping harmful chemicals into the environment.

The Environmental Working Group (EWG) has studied the current state of our world in great detail and has discovered that before a child is even born they already have approximately 287 toxins in their blood and tissues. These results came from 10 newborns whose parents gave permission to have their toxins measured at birth.

The results of this study indicate that an average of 200 chemicals was found in each newborn. Of the toxins tested, 47 were consumer ingredients such as cosmetics, 212 were industrial and pesticide byproducts. In this study, only around 400 total chemicals were actually tested for - thousands of others may have been found if larger parameters were used.

Many of the toxins measured in the newborns included plastics, flame retardants, and other chemicals that disrupt brain function, IQ, hormones, and the nervous system of the child. Some of the toxins observed like DDT, have actually been banned since 1972 (over 3 decades ago), but are still being measured in laboratory samples. Certain chemicals never fully degrade in the environment.

So, there is not question about it, the environment we live in is toxic. All of us have disease-creating toxins inside of our bodies, the question boils down to which ones and how much.

But there are some solutions: lab tests and detoxification.

Dr. Sharon Goldberg, an internal medicine physician and professor gives her testimony at Michigan's 5G Small Cell Tower Legislation Hearing on October 4, 2018 regarding the dangers of electromagnetic radiation.

She says: "Wireless radiation has biological effects. Period. This is no longer a subject for debate when you look at PubMed and the peer-review literature. These effects are seen in all life forms; plants, animals, insects, microbes. In humans we have clear evidence of cancer now; there is no question. We have evidence of DNA damage, cardiomyopathy, which is the precursor of congestive heart failure, neuropsychiatric effects... 5G is an untested application of a technology that we know is harmful; we know it from the science. In academics this is called human subjects research."

Transcript:
"You hear all this talk about blue light being harmful, but do you know what blue light actually is?

Blue light is one of the types of light that form the white light we get from the Sun. Together with
red, orange, yellow, green, violet, and indigo. This is called the electromagnetic (EM) spectrum of visible light. At the end we have the UV light, which can't actually be seen by the human eye. The energy of these waves increases as we go towards the end, which makes blue lights one of the highest intensity types of visible light.

Light is made of EM waves that emit energy and it's this energy that we perceive as light. These waves come in different wavelengths, which means we get different colors of light. These are the different colors of light that we can perceive from the EM spectrum. So blue light is actually everywhere; it's in the light that travels from the Sun all the way to the Earth.

Because the wavelength of blue light is so small they collide with air molecules a lot more than any other color and they get scattered everywhere - that's actually what makes the sky blue. That's why your body uses this blue light from the Sun to make the difference between day and night and
regulate your sleep cycle. But our eyes natural filters barely provide us with enough protection from blue light on a particularly sunny day.

The blue light from your devices is even worse. LED devices emit much stronger blue light than we get from the Sun. Spending hours staring at a screen can cause eye damage and fatigue. This is due to most lower energy waves being absorbed by the cornea, the eyes outer membrane. Blue light goes straight through due to its high energy and slowly deteriorates the retina. And because our brains use blue light to differentiate between day and night and boost alertness, spending time on your phone tablet or laptop late at night fools your body into thinking it should keep you awake. It's all of them - phones, tablets, any gadget with an illuminated display. They all use blue LEDs because they are energy efficient and cheaper to produce, but your body disagrees.

Basically, what keeps you awake and alert during the day can severely affect the quality of your sleep at night. Blue light has also been shown to suppress secretion of melatonin, a hormone that is produced at night and helps your body prepare for sleep. Melatonin isn't just linked to poor
sleep, scientists have also managed to find a correlation between melatonin deprivation and conditions like cancer, diabetes and clinical depression. Do you still think your tablet is that harmless?

Well there is a way you can prevent it. Scientists have now designed special screen protectors that stop the blue light from reaching your eyes and causing damage to your retina at a microscopic level. This glass has tiny ridges that block blue waves and let the other less harmful light go through. These glasses can block up to 60% of blue light and 99% of UV rays. People have reported that their sleeping patterns were significantly improved after only a few days. There was also significant reduction in eye strain and headaches. Or you can go for full protection and buy eyewear that you can use for all blue light-emitting devices: computers, TV laptops, or your phone. These work in a slightly different way - the protectors as the yellow, absorbs rather than blocks blue and UV light, and lets other types of light go through.

No matter what you do, whether its buying a protective glass or reducing the time you spend on your device make sure you stay on the lookout for the unseen damage that your day-to-day gadgets can do to your health."

Key benefits of red light include:

1. Minimal Melatonin Suppression: Studies show that exposure to red light has little to no impact on melatonin levels, allowing the body to maintain its natural sleep-wake cycle.
2. Reduced Eye Strain: Red light is gentler on the eyes compared to the harsh glare of blue or white light, especially in low-light conditions.
3. Natural Ambiance: The warm glow of red incandescent bulbs mimics the light of a natural sunset, signaling the brain to prepare for rest.

Applications of Red Incandescent Lighting

• Evening Lighting: Replace bright white or blue-tinged LED or CFL bulbs with red incandescent bulbs in bedrooms, living rooms, and bathrooms.
• Nighttime Usage: For those who need to wake during the night, red light provides sufficient illumination without disrupting sleep.
• Technology Filters: Pair red lighting with blue-light filtering settings on electronic devices to further minimize blue light exposure.

​Several studies support the use of red light for circadian health:

• Melatonin Preservation: Research indicates that red light exposure does not suppress melatonin production, making it ideal for nighttime environments. 
• Minimized Disruption: Comparisons between red and blue light exposure highlight red light’s negligible impact on the SCN and circadian timing.

When selecting red incandescent light bulbs:

• Opt for bulbs labeled as "true red" to ensure they emit light solely in the red spectrum.
• Choose low-wattage bulbs (15-25 watts) to create a soothing ambiance without excessive brightness.
• Confirm the absence of blue or white light leakage by testing in a dark room.

Electromagnetic radiation. In this video Dr. Mercola helps you take control of your health by sharing information about electromagnetic radiation, the dangers, and what can you
do to protect yourself.

When it comes to EMF (electromagnetic frequencies), there are two general categories: native (or natural) and non-native (or artificial). Sunlight is an example of of native EMF and it actually encourages and stimulates health. Artificial EMF almost always is going to push you in the wrong direction not always but almost always. This includes artificial light such as LED and fluorescent lights as well as microwave emitters such as microwave ovens, cellphones and celltowers, and Wi-Fi routers.

New research indicates that chronic exposure to high levels of non-native microwave radiation will sabotage your mitochondria and may result in a wide-variety of chronic diseases, such as cancer. Non-native EMFs disrupts mitochondrial function through a reactive nitrogen species called peroxy nitrate resulting more hydroxyl free radicals.

More than 2000 references on the biological responses to radio frequency (RF) and microwave radiation, published up to June 1971, have been well-documented by the U.S. Naval Medical Research Institute. Devices that emit RF and microwave radiation include, but is not limited to, cellphones, two-way radios, Wi-Fi routers, cellphone towers, smart watches, bluetooth devices, Smart meters, cordless cell phone base stations, wireless baby monitors, microwave ovens, and any WiFi-connected smart devices that receives and transmits data. Particular attention has been paid to the effects on man of non-ionizing radiation at these frequencies.

Reported Biological Phenomena (*Effects') and Some Clinical Manifestations Attributed to Microwave and Radio-Frequency Radiation

A. Heating of Organs*
(Applications: Diathermy, Electrosurgery, Electro-coagulation, Electrodesiccation, Electrotomy)

1. Whole Body (temperature regulation defects), Hyperpyrexia
2. Skin
3. Bone and Bone marrow
4. (a) Lens of Lye (cataractous lesions - due to the avascular nature of the lens which prevents adequate heat dissipation (b) Corneal damage also possible at extremely high frequencies
5. Cenitalia (tubular degeneration of testicles)
6. Brain
7. Sinuses
8. Metal Implants (burns near hip pins, etc.)

The effects are generally reversible except for 4a.

B.  Changes in physiologic function

1. Striated Muscle Contraction
2. Alteration of Diameter of Blood Vessels (increased Vascular elasticity, Dilation
3. Changes in Oxidative Processes in Tissues and Organs
4. Liver Enlargement
5. Altered Sensitivity to Drug Stimuli
6. Decreased Spermatogenesis (decreased fertility, to sterility)
7. Altered Sex Ratio of Births (more girls!)
8. Altered Menstrual Activity
9. Altered Fetal Development
10. Increased Lactation in Nursing Mothers
11. Reduction in Piuresis (Na+ excretion, via urine output)
12. Altered Renal function (decreased filtration in tubules)
13. Changes in Conditioned Reflexes
14. Increased Electrical Resistance of Skin
15. Changes in the Structure of Skin receptors of the (a) Digestive, and  (b) Blood Carrying Systems
16. Altered BIood Flow Rate
17. Alterations In the Biocurrents (EEG?) of the Cerebral Cortex (in animals)
18. Changes In the Rate of Clearance of Tagged Ions from Tissue
19. Reversible Structural Changes In the Cerebral Cortex and the Diencephalon
20. Electrocardiographic (EKG) Changes
21. Alterations In Sensitivity to Light, Sound, and Olfactory Stimuli
22. Functional (a) and Pathological (b) Changes in the Eyes: (a) decrease in size of blind spot, altered color recognition, changes in intraocular pressure, lacrimation, trembling of eye-lids; (b) less opacity and coagulation, altered tissue respiration, and altered reduction-oxidation processes
23. Myocardial Necrosis
24. Hemorrhage in Lungs, Liver, Gut, and Brain (At Fatal Levels of Radiation)
25. Generalized Degeneration of all Body Tissue of Radiation (At Fatal Levels of Radiation)
26. Loss of Anatomical Parts
27. Death
28. Dehydration
29. Altered Rate of Calcification of Certain Tissue

C.  Central Nervous System Effects

1. Headaches
2. Insomnia
3. Restlessness (Awake and During Sleep)
4. Electroencephalographic (EEG) Changes
5. Cranial Nerve Disorders
6. Pyramidal Tract Lesions
7. Conditioned Reflex Disorders
8. Vagomimetic Action of the Heart; Sympaticomimetic Action
9. Seizures, Convulsions

D.  Autonomic Nervous System Effects

1. Degenerative Disorders (e.g., alteration of heart rhythm)
2. Fatigue
3. Structural Alterations in the Synapses of the Vagus Nerve
4. Stimulation of Parasympathetic Nervous System (Bradycardia), and Inhibition of  the Sympathetic Nervous System

E.  Peripheral Nervous System Effects

1. Effects on Locomotor Nerves

F. Psychological Disorders ("Human Behavioral Studies") - the so-called "Psychophysiologic (and Psychosomatic) Responses"

1. Neurasthenia - (general "bad" feeling)
2. Depression
3. Impotence
4. Anxiety
5. Lack of Concentration
6. Hypochondria
7. Dizziness
8. Hallucinations
9. Sleepiness
10. Insomnia
11. Increased Irritability
12. Decreased Appetite
13. Loss of Memory
14. Scalp Sensations
15. Increased Fatigability
16. Chest pain
17. Tremor of the hand

G. Behavioral Changes (Animal)

1. Reflexive, Operant, Avoidance, and Discrimination Behaviors

H. Blood Disorders
changes in:

1. Blood and Bone Marrow
2. Phagocytic (polymorphs) and Bactericidal Functions
3. Hemolysis Rate (increase), (a shortened lifespan of cells)
4. Sedimentation Rate (increase), due to changes in serum concentration levels or amount of fibrinogen. (?))
5. Number of Erythrocytes (decrease), also number of lymphocytes
6. Blood Glucose Concentration (increase)
7. Blood Histamine Content
8. Cholesterol and Lipids
9. Gamma (also alpha and beta) Globulin, and Total Protein Concetration
10. Number of Eosinophils
11. Albumin/Globulin Ratio (decrease)
12. Hemopoiesis (rate of formation of blood corpuscles)
13. Leukopenia (increase in number of white cells), and Leukocytosis
14. Reticulocytosis

I. Vascular Disorders

1. Thrombosis
2. Hypertension

J. Enzyme and Other Biochemical Changes
Changes in activity of:

1. Cholinesterase
2. Phosphatase
3. Transaminase
4. Amylase
5. Carboxydismutase
6. Protein Denaturation
7. Toxin, Fungus, and Virus Inactivation (at high radiation dose levels), Bacteriostatic  Effect
8. Tissue Cultures Killed
9. Alteration In Rate of Cell Division
10. Increased Concentration of RNA in Lymphocytes, and Decreased Concentration in  Brain.  Liver,  and  Spleen
11. Changes in Pyruvic Acid, Lactic Acid, and Creatinine Excretions
12. Change in Concentration of Glycogen in Liver (Hlyperglycemia)
13. Alteration in Concentration of 17- Ketosteroids in Urine

K. Metabolic Disorders

1. Glycosuria (sugar in urine; related with blood sugar?)
2. Increase in Urinary Phenol (derivatives? DOPA?)
3. Alteration of rate of metabolic Enzymatic Processes
4. Altered Carbohydrate Metabolism

L. Gastro-Intestinal Disorders

1. Anorexia (loss of appetite)
2. Epigastric Pain
3. Constipation
4. Altered Secretion of Stomach "Digestive Juices"

M. Endocrine  Gland  Changes

1. Altered Pituitary Function
2. Hyperthyroidism
3. Thyroid Enlargement
4. Increased Uptake of Radioactive Iodine by Thyroid Gland
5. Altered Adrenal Cortex Activity
6. Decreased Corticosteroids in Blood
7. Decreased Glucocorticoidal Activity
8. Hypogonadism (usually decreased testosterone production)

N. Histological Changes

1. Changes in Tubular Epithelium of Testicles
2. Gross Changes

O. Genetic and Chromosomal Changes

1. Chromosome Aberrations (e.g., linear shortening, pseudochiasm, diploid structures, amitotic division, bridging, "sticky" chromosomes, irregularities in chromosomal envelope)
2. Mutations
3. Mongolism
4. Somatic Alterations (changes in cell not involving nucleus or chromosomes, cellular transformation)
5. Neoplastic Diseases (e.g*, tumors)

P. Pearl Chain Effect (Intracellular orientation of subcellular particles, and orientation of cellular and other (non-biologic) particles) Also, orientation of animals, birds, and fish in electromagnetic fields

Q. Miscellaneous Effects

1. Sparking between dental fillings
2. Peculiar metallic taste in mouth
3. Changes in Optical Activity of Colloidal Solutions
4. Treatment for Syphilis, Poliomyelitis, Skin Diseases
5. Loss of Hair
6. Brittleness of Hair
7. Sensations of Buzzing Vibrations, Pulsations, and Tickling About the Head and Ears
8. Copious Perspiration, Salivation, and Protrusion of Tongue
9. Changes in the Operation of Implanted Cardiac Pacemakers
10. Changes in Circadian Rhythms

Glaser, Z. (1971). Bibliography of reported biological phenomena ('effects') and clinical manifestations attributed to microwave and radio-frequency radiation. Navel Medical Research Institute. https://archive.org/details/DTIC_AD0750271/mode/2up?view=theater​